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Butyrate produced by commensal bacteria potentiates phorbol esters induced AP-1 response in human intestinal epithelial cells

Moreover, autophagy, especially selective autophagy (e.g., lipophagy, ferritinophagy, clockophagy, and mitophagy), promotes lipid catabolism or lipid peroxidation-induced ferroptosis through the degradation of various substances within the cell

Consequently, the nutritional approach must be highly nuanced, balancing the need to reduce systemic inflammation with the necessity of minimizing the dietary histamine burden

doi: 10.1080/095530099139449

Single enzymes affect different hallmarks of ferroptosis, e.g., Prx6 can provide GPX4 with selenium, directly reduce lipid hydroperoxides, or regulate the MUFA/PUFA ratio

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