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Sequentially, Vit-C-mediated restoration of TET, also when mutated, enables the re-expression of tumour-suppressor genes in cancer cells [2, 3, 105, 174]

However, sustained high doses, overdose, fasting, alcohol use, or pre-existing liver disease can overwhelm glutathione reserves, increasing risk of hepatotoxicity and oxidative stress

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7 LET7 TPR and HOP1 stabilize SUMM2 but not CRCK3

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