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The bacteriostatic agent effectively prevents bacterial growth, hence the term "bacteriostatic"

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The receptor profiles are different (dual versus triple agonist), the potency per milligram is different, and the side effect profiles are different

These studies are mechanistically aligned with the VEGFR2/eNOS in vitro findings but rely on the same originating laboratory and rodent model system, and independent replication outside that network is sparse

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