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The increased lipid ROS brought about by SOD1 G93A protein coupled with a compromised anti-ferroptosis defense system would render motor neurons extremely vulnerable to ferroptosis

For instance, the CRISPR-PASS system corrected a nonsense mutation in the XPC gene, and achieved protein restoration in patient-derived cells [189]

However, genetic studies cannot provide the same fine-tuning and evaluation of dose-dependency and dosage scheduling that is possible with pharmacological agents

The Dichloroacetate Dilemma: Environmental Hazard versus Therapeutic Goldmine-Both or Neither

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