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Also, in the late phase of M1 macrophage polarization, lactate-derived lactylation of histone lysine residues as observed in conditions of hypoxia and bacterial challenges served as an epigenetic modification that directly stimulated the expression of homeostatic genes that have been traditionally associated with M2-like macrophages (Zhang et al., 2019)

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Additionally, BER prevents the activation of the LRR, NACHT, and PYD domain-containing protein 3 (NLRP3) inflammasome during PD-related inflammation and restores autophagy activity that safeguards dopamine neurons [24]

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