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doi: 10.1007/s10545-017-0063-1

Dec 2006;72(15):1378-82

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Hence, inhibiting renal PGHS-2 by NSAIDs could increase asymmetric dimethyl arginine (ADMA), an endogenous eNOS inhibitor, which eventually could block eNOS, minimizing the protective effect of NO and thereby increasing hypertension, atherosclerosis and thrombosis [98], [166]

This occurs through the activation of PKA signaling components which are recruited to the mitochondrial outer membrane by the anchoring protein AKAP10