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Transcriptomic analyses have attempted to classify GB into subtypes that could predict treatment response 1,2,3 , and a recent study that used a pathway-based classification defined metabolism-associated subtypes with distinct therapeutic vulnerabilities

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Activation of oncogenes such as RAS and MYC, along with metabolic reprogramming phenomena like the Warburg effect, further amplify intracellular ROS generation, establishing a reinforcing cycle that perpetuates tumor development [34]

The gut as a potential trigger of exercise-induced inflammatory responses