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Interestingly, mitochondrial dysfunction has been demonstrated in animal models induced by exogenous toxicants such as the propionic acid adult rodent model (39, 40) and the prenatal valproic acid exposure rodent model (41), as well as in genetic animal models of ASD, including the Rett syndrome (28), phosphatase and tensin homolog gene haploinsufficiency (42), and Angelman syndrome (43) rodent models of ASD

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