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Although the signaling pathways of GDNF family in cavernous nerve regeneration have not been completely clarified, Wanigasekara and Keast (2005) have proven that NTN initiates axonal growth of parasympathetic neurons of the MPG through the PI3K pathway and regulates microtubule assembly through the MAPK and Src kinase pathways

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In the present study, no differences in power output between trials indicates a lack of sufficient change in skeletal muscle content of L-carnitine

The accumulation of methylmalonic acid (MMA) and Hcy because of these disruptions leads to oxidative damage and impaired methylation