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The animal science is real, but calling it 'proven' is a stretch when 99.7% of the research is in rats

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Several papers showed that the excess amount of ROS is involved in the development and progression of chronic diseases, including the pathogenesis of insulin resistance and type-2 diabetes

Discussions and perspectives The protective role of UA in neurodegenerative diseases is supported by evidence from animal and in vitro models

Recovery is shown by restoration of transepithelial electrical resistance (TEER) to the starting value after washout, and by relocalization of the tight junction proteins claudin and occludin, meaning that these proteins return to the cell borders in microscopy images