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To determine whether increases in intracellular GSH directly enhance expression of T2-signature proteins in HAECs, we lowered intracellular GSH levels by inhibiting SLC7A11 using the chemical inhibitor erastin (16)

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Anxiety traits and CYP2D6 variants: I mentioned above that CYP2D6 is involved in[ref][ref] The metabolism of tyrosine to dopamine

Third, lack of HO-1 expression in bone marrow-derived cells resulted in atherosclerotic plaques with a larger inflammatory component, suggestive of greater vulnerability for rupture (Orozco et al., 2007)

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