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In our cell model, SOD1 G93A induced only modest toxicity to NSC-34 cells, which may be the reason why we found ferroptosis was largely responsible for death induced by SOD1 G93A in this model

This disruption promotes TG accumulation and decreases HDL-C synthesis, contributing to obesity-related dyslipidemia (Klop et al., 2013) (Figure 2)

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In a postmortem study of 13 patients with idiopathic PD and three with genetic PD (compared with 12 controls), all PD patients showed IgG (mostly IgG1) but no IgM binding to dopaminergic neurons, while Lewy bodies were strongly immunolabelled with IgG (94)

Nickel (Ni)- and iron-zinc (Fe/Zn) containing isozymes have also been identified in several bacteria [17, 18]

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