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In glutamate-exposed HT22 cells, -Oryzanol improved m , alleviated GSH depletion, and reduced the expression level of CAMKII, thereby protecting neurons from glutamate excitotoxicity [187]

Our findings are consistent with earlier studies demonstrating that low-dose radiation priming can trigger an adaptive response: in human A549 cells, a 0.5 Gy pre-treatment decreased apoptosis and altered inflammatory and antioxidant markers 62 in human peripheral blood mononuclear cells, gamma radiation triggered antioxidant pathways such as Nrf2 56

Similarly, given the sensitivity of ACSL4 as a promotor of lipid peroxidation, an initial study demonstrated that targeting inhibition of ACSL4 via dexmedetomidine protect kidneys against IRI-induced kidney injury [249] (Fig

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