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The Keap1/Nrf2/ARE pathway is a key mediator of cytoprotective responses to oxidative and electrophilic stressors

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Alternatively, activation of NFB in the presence of excess hydrogen peroxide has been shown to enhance the duration and intensity of the NFB activation

SOD1 (copper/zinc superoxide dismutase) deficiency drives amyloid beta protein oligomerization and memory loss in mouse model of Alzheimer disease

Anti-RBD antibodies may also block the second step of viral entry: membrane fusion

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