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Exposure to paraquat can induce pulmonary toxicity which has been demonstrated in previous studies that showed retention of paraquat occurs higher in the lung than in other organs (Sharp et al., 1972)

Frataxin deficiency disrupts mitochondrial respiration and pulmonary endothelial cell function

The compounds ability to cross the blood-brain barrier and accumulate in mitochondria-rich tissues has made it an interesting candidate for therapeutic applications targeting mitochondrial health Figure 3 [71]

K.UpadhyayS

Nanoparticles for vaccine and gene therapy: overcoming the barriers to nucleic acid delivery

The alleviation of the abovementioned pathologies caused by arsenic toxicity may prevent the incidence and progression of a wide range of cardiovascular diseases