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interaction of haloacetonitriles with glutathione and glutathione-s-transferase Induce Structure-Related Cellular Toxicity Through Distinct Proteome Thiol Reaction Mechanisms Oxidative injury induced by drinking

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such off-label use carries risk and should not be undertaken without medical supervision

interaction of haloacetonitriles with glutathione and glutathione-s-transferase Induce Structure-Related Cellular Toxicity Through Distinct Proteome Thiol Reaction Mechanisms Oxidative injury induced by drinking

Song X, Zhuang W, Cui H, Liu M, Gao T, Li A, et al

interaction of haloacetonitriles with glutathione and glutathione-s-transferase Induce Structure-Related Cellular Toxicity Through Distinct Proteome Thiol Reaction Mechanisms Oxidative injury induced by drinking

H.PrangeH.ReimersC

interaction of haloacetonitriles with glutathione and glutathione-s-transferase Induce Structure-Related Cellular Toxicity Through Distinct Proteome Thiol Reaction Mechanisms Oxidative injury induced by drinking

However, drug-induced parkinsonism is a recurrent undesirable effect of several drugs that impede dopaminergic function particularly in the basal ganglia, 29,30 including calcium antagonists, orthopramides, and substituted benzamides

interaction of haloacetonitriles with glutathione and glutathione-s-transferase Induce Structure-Related Cellular Toxicity Through Distinct Proteome Thiol Reaction Mechanisms Oxidative injury induced by drinking

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interaction of haloacetonitriles with glutathione and glutathione-s-transferase Induce Structure-Related Cellular Toxicity Through Distinct Proteome Thiol Reaction Mechanisms Oxidative injury induced by drinking

doi: 10.1055/s-0043-1770985 55

interaction of haloacetonitriles with glutathione and glutathione-s-transferase Induce Structure-Related Cellular Toxicity Through Distinct Proteome Thiol Reaction Mechanisms Oxidative injury induced by drinking

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