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Inhibition of GSH and Trx metabolism with buthionine sulfoximine (BSO) and auranofin (AUR), respectively, induced significant decreases in clonogenic survival compared to either drug alone in FaDu, Cal-27 and SCC-25 HNSCC cells in vitro and in vivo in Cal-27 xenografts

Our findings reveal a bacterial-host metabolic axis that links oxidative stress, iron homeostasis, and longevity in C

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After a month, TMAO levels for red meat eaters tripled compared to those consuming white meat or non-meat protein sources

Tissue regeneration mechanisms
