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In the context of high-grade NB, a defining characteristic is the overexpression of the oncogene MYCN [155]
Additionally, UCMSCs-Exos enhance tight junction protein expression via the miR-501-5p/myosin light chain kinase (MLCK) and miR-149/endothelin (ET-1) axes, thereby stabilizing BSCB MyD88 function and fostering a favorable microenvironment for neural repair (5256)
PMID: 25052051
doi: 10.1186/s12889-018-6062-9
Georges University School of Medicine and completed his residency training at Memorial Hermann Southwest Hospital
