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The wild type, but not a mutant form of -synuclein is degradated by CMA, suggesting that this mechanism is important in the formation of -synuclein aggregates during PD [114]

These products are released from the adipocyte into the bloodstream, where fatty acids bind to albumin for transport to tissues that oxidize them for energy

Materials and Methods 2.1

Neurochem Res 41(12):6472 Piccinin E, Sardanelli AM, Seibel P, Moschetta A, Cocco T, Villani G (2021) PGC-1s in the spotlight with Parkinsons Disease

The main reason for paracetamol to be the commonest drug responsible for self-poisoning is that it is readily available [36]

Acquired amino acid deficiencies: a focus on arginine and glutamine