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Taken together, our data suggest that the acetyl-CoA buildup secondary to FASN deletion inhibits ATP citrate lyase and induces an accumulation of citrate/isocitrate

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We show that inhibition of ATP synthesis caused a marked intracellular accumulation of pyroglutamate, reflecting decreased GSH synthesis

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This activation scavenges ROS, reduces the consumption of metabolic substrates like succinate, and mitigates excessive ROS production and lipid peroxidation, thereby exerting an antioxidant effect on the myocardium (Jiang et al., 2019)