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MOTS-C attacks inflammation directly by suppressing the expression of TNF-alpha, IL-6, and IL-1beta in multiple tissue types

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Pain Processing Mechanisms Central Sensitization (via mGlu2 receptor upregulation)[4][5] Neuroinflammation (modulates glial activation)[12][13] Mitochondrial Dysfunction (enhances ATP production, -oxidation)[2][13] Oxidative Stress (direct antioxidant activity)[2][13] Systemic Inflammation limited direct evidence SUMMARY OVERVIEW CLINICAL PEARLS 1

BLOC-1 is required for cargo-specific sorting from vacuolar early endosomes toward lysosome-related organelles