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Ketones have the potential to activate these signaling pathways via G-protein receptor binding or through MCT-mediated transport into the cell 43

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sativa reduced the histologically monitored cheek mucosal damage in mucositis (Lotfy and Zayed, 2009)

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As a result, both SahH-T216A and SahH-T216A/T219A/T220A/T221A mutants were hyperphosphorylated (Fig