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glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport reduced glutathione and breast cancer

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The datasets used and/or analyzed during the present study are also available from the corresponding author on reasonable request

glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport reduced glutathione and breast cancer

Antibiotic stewardship in the intensive care unit

glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport reduced glutathione and breast cancer

[DOI] [PubMed] [Google Scholar] 21.Tolosa de Talamoni N, Marchionatti A, Baudino V, Alisio A

glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport reduced glutathione and breast cancer

Green MF, Scott JW, Steel R, Oakhill JS, Kemp BE, Means AR

glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport reduced glutathione and breast cancer

Rev ., 24 (137), 451461

glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport reduced glutathione and breast cancer

The overexpression of miR-21-3p correlates with the inhibition of critical neuronal and synaptic genes such as DLGAP1 , which decreased the expression of Protocadherin-19 ( PCDH19 ), associated with cognitive impairment

glutathione colon cancer DLAT sustains redox homeostasis and prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport reduced glutathione and breast cancer

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