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Conjugation site heterogeneity causes variable electrostatic properties in fc conjugates

Moreover, mitochondrial dysfunction and related oxidative stress can activate the innate branch of the immune system, leading to the release of pro-inflammatory cytokines influencing various depression-related pathophysiological mechanisms: monoaminergic neurotransmission disruption, tryptophan degradation toward neurotoxic catabolites, glutamate-related excitotoxicity, decreased neurotrophic factors and alterations in the hypothalamic-pituitary-adrenal axis [3, 33, 59]

Mass spectrometry-based metabolomics analysis and stable isotope tracing To measure oxPPP and non-oxPPP activity, proliferating chondrocytes were cultured for 24 h in glucose-free DMEM supplemented with 10% dialysed FBS and 5.5 mM [1,2]- 13 C 2 -glucose (Cambridge Isotope Laboratories)

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