glutathione peroxidase deficiency hepatic Antioxidant effects of Se-glutathione in alcoholic liver disease The interplay between glutathione peroxidase

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However, when ERS persists or becomes overactivated, it surpasses cellular adaptive thresholds and subsequently modulates various forms of programmed cell death (PCD), including apoptosis, autophagy, pyroptosis, and ferroptosis, thereby contributing to tumorigenesis, therapy resistance, and immune microenvironment remodeling [4, 5]

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In the case of thiol groups, the cysteinyl residues in several HMM-SH molecules have no significant functional or catalytic role

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