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The mechanism of these strains has demonstrated hypocholesterolemic effects through their ability to reduce serum levels of triglycerides, low-density lipoprotein (LDL), and high-density lipoprotein (HDL) (414)
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The peptides work through complementary mechanisms without competition
We speculate that downregulation of HIF-1 via the mTOR1 pathway or alleviation of the hypoxic microenvironment may attenuate the Warburg effect, impede T cell differentiation, inhibit glycolytic metabolism, and modulate inflammatory and immune responses in AID patients such as SLE
The same is true for NSAIDs (ketoconazole and lysophylline), statins and albuterol, for example [45]
