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Description
McConnachie, A
CHAC1 emerges as a key stress-responsive effector, transcriptionally upregulated via the ATF4-CHOP axis during endoplasmic reticulum stress and amino acid deprivation
Moreover, loss of function of PINK1, disrupts mitochondrial autophagy and contributes to an increase in ROS levels (Requejo-Aguilar et al., 2014)
Published online 2013
S., Hatabu A., Takagi T., Ueda M., Ikeda K
The gene expression of the gene of interest was normalized to -actin , and Gapdh
