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Dysregulation of the maternal immune system during PE leads to overstimulation of the complement system as a compensatory mechanism (82), with recruitment of phagocytic cells and neutrophils to the site of stimulation (83)

doi: 10.3389/fpls.2020.575829 32 GhatakA.ChaturvediP.WeckwerthW

Catecholamines acting on endothelial cell adhesion molecules and chemokines produce a similar dissociation in the modulation of the innate and adaptive immune system during the dark-light cycle

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Although lipid peroxidation products (e.g., 4-HNE, MDA) and systemic iron parameters are altered in bodily fluids of AD patients (e.g., cerebrospinal fluid, blood), these markers lack disease and cell death modality specificity, as similar changes can occur in other neurodegenerative disorders or systemic pathological conditions