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For example, inhibiting Ras activity under specific conditions, such as uniform membrane distribution of RasGAP or rear-targeting, can enhance posterior myosin contractility, thereby accelerating cell polarization and migration (321)

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The G6PD deficiency is produced by different genetic mechanisms such as deletions, precise mutations and substitutions that affect the transcription, process or primary enzyme structure, which functionally results in a decrease of the enzymatic activity or loss of affinity by the substrate