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The Mitochondrial-Encoded Peptide MOTS-c Translocates to the Nucleus to Regulate Nuclear Gene Expression in Response to Metabolic Stress

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Purpose A patient was identified with severe metabolic acidosis, a high anion gap and 5-oxoproline accumulation, probably caused by the simultaneous use of paracetamol (acetaminophen) and flucloxacillin

Within research contexts, BPC-157 and TB-500 are often discussed together due to their complementary roles in studies examining recovery mechanisms, tissue integrity, and cellular communication

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