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glutathione alcoholic hepatitis disulfide sensitizes hepatocytes to TNFα-mediated cytotoxicity via IKK-β S-glutathionylation: a potential mechanism underlying non- fatty liver disease jcdr-7-1568-g004.jpg

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glutathione alcoholic hepatitis disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non- fatty liver disease jcdr-7-1568-g004.jpg

doi: 10.1017/S1368980013000475

glutathione alcoholic hepatitis disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non- fatty liver disease jcdr-7-1568-g004.jpg

[DOI] [PMC free article] [PubMed] [Google Scholar] Cummings, J

glutathione alcoholic hepatitis disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non- fatty liver disease jcdr-7-1568-g004.jpg

doi:10.1111/nbu.12712 Mathew AR, Di Matteo G, La Rosa P, et al

glutathione alcoholic hepatitis disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non- fatty liver disease jcdr-7-1568-g004.jpg

Maternal riboflavin deficiency, resulting in transient neonatal-onset glutaric aciduria Type 2, is caused by a microdeletion in the riboflavin transporter gene GPR172B

glutathione alcoholic hepatitis disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non- fatty liver disease jcdr-7-1568-g004.jpg

Petersburg Institute of Bioregulation and Gerontology evaluated the effect of Epithalon on telomerase activity and telomere length in cultured human fibroblasts

glutathione alcoholic hepatitis disulfide sensitizes hepatocytes to TNF-mediated cytotoxicity via IKK- S-glutathionylation: a potential mechanism underlying non- fatty liver disease jcdr-7-1568-g004.jpg

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