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The Pressure Dependence of Hydrophobic Interactions Is Consistent With the Observed Pressure Denaturation of Proteins

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As explained in two excellent recent reviews [18, 88] and illustrated in Figure 4, the hypomagnesaemia is due to the induction of a loss of function mutation in the epithelial Mg2+ channel TRPM6 (transient receptor potential cation channel, subfamily M, member 6), one of the main tubular magnesium channels in the distal collecting tubular cells

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