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As a result, the primary source for synaptic calcium entry shifts from NMDARs in nave animals to GluA2-lacking AMPARs after a dose of an addictive drug

[DOI] [PubMed] [Google Scholar] 120.Ali-Osman F., Brunner J.M., Kutluk T.M., Hess K

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Ischemic wounds treated with GHK-Cu show significantly decreased MMP2 and MMP9 concentrations compared to controls, accompanied by faster healing and reduced TNF-