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Description
Glucagon signalling in hepatocytes suppresses intermediaries in fatty acid synthesis and prompts catabolism of FFAs, leading them to beta-oxidation and degradation, thereby diverting them from storage as triglycerides and eventual release in the form of VLDL [72]

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Steele et al., 2015)

"If we saw obesity as a disease, and we're both focused on preventing it but once it happens treating it and treating it as a disease, I think we're going to see an entirely different approach," he said
