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In addition, PPAR interacts with a consensus DR-1 element located about 50 bp further upstream, which also binds HNF4 and COUP-TF 8 , and PPAR binding at this position represses SHBG transcription 9

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Pharmacological strategies that block PERK, NF-B, or NLRP3 have reduced TMAO induced inflammation in preclinical models, indicating their translatable potential