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and to Nestle Institute of Health Sciences for funding support

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This allows the system to reset and maintain sensitivity for future studies

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194 This result was consistent with the accumulation of CSA in patient-derived low-grade glioma and with the intra-tumoural expression of CDO1, 76 since the decreased level of cysteine can be due to the overexpression and activity of CDO1 found in high-grade glioblastomas

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Conversely, loss of Notch activity results in upregulation of transcription factors such as Ascl1/Mash1, Neurog2, and NeuroD driving neuronal commitment

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