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Another mechanism involves the oxidative inactivation of cyclic guanosine monophosphate (cGMP), which mediates the effects of NO on sleep homeostasis, and the upregulation of NF-kB by inducible nitric oxide synthase (iNOS) from activated microglia, significantly influencing sleep homeostasis (Cespuglio et al
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Hiby N, Bjarnsholt T, Moser C, Bassi GL, Coenye T, Donelli G, et al
In a current study, Sokolowski, Kucharska-Lusina (101) showed reduced gene expression and protein concentration of GDNF in the plasma of MS-patients, indicating impaired neuroprotection
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It usually resolves within a few hours or days before the next oxaliplatin cycle [3]
