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Discussion The treatment of RA is a complex process aimed at protecting the function of joints and muscles as much as possible, ultimately achieving the goal of complete remission or reducing disease activity

Moreover, L-Carnitine suppressed genes responsible for protein degradation in skeletal muscle [17] and decreased muscle RING-finger protein-1 ( MuRF1 ) and ubiquitin-protein conjugates, involved in protein catabolism, as well as increases the levels of IGF-1 and Akt1, two upstream elements in the mammalian target of rapamycin (mTOR), the main driver of protein anabolism [18]

Using neuroproteomic approaches, we can also better characterize brain region-specific alterations that contribute to neural dysfunction and cognitive impairments with conditions ranging from normal aging to neurodegenerative diseases

ER promotes A degradation via the modulation of autophagy

SK: Formal Analysis, Investigation, Methodology, Validation, Writing review & editing

In males, early testosterone depletion has been identified as a risk factor for early-onset AD, with lower testosterone levels frequently linked to cognitive impairment [180]