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207 Ritacca, A

Disruption of this network may lead to systemic iron overload and contribute to pathological processes such as oxidative stress, inflammation, and ferroptosis, ultimately promoting lung injury and the progression of pulmonary fibrosis

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In contrast, it has been reported that in the neurons of mice with Fragile X syndrome (FXS), ATP5G1, the c subunit of F 0 -ATP synthase, is abnormally upregulated

mTORC1 Pathway The mechanistic target of rapamycin complex 1 (mTORC1) pathway functions as the primary regulator of cellular metabolism based on nutrient availability

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