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10.1016/s0028-3908(00)00069-1 109 RuelJ.WangJ.RebillardG.EybalinM.LloydR.PujolR.et al
Inducible NOS (NOS2) and neuronal NOS (NOS1) are involved in neurotoxicity, leading to excitotoxicity cascades, inflammation, apoptosis, and deterioration to the primary brain injury through the release of free radicals and production of nitrates, which directly damage mitochondrial enzymes and genetic material (Sims and Anderson, 2002
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Mitochondrial ROS inhibitor Mito-TEMPO reverted the process of senescence and decreased the p53/p21 expression of BM-MSCs [187]
Once at the tissue level, E2 exerts its actions through estrogen receptors and (ER and ER), as well as membrane-associated receptors mediating rapid non-genomic signaling [30,31]
In contrast, patients on CYP3A4-inducing drugs who also take GCs may require greater GC doses to achieve the intended treatment effect
