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Description
No human studies bridge that gap, so the muscle-repair evidence should be attributed to IGF-1 biology broadly, not to IGF-1 LR3 specifically
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[DOI] [PMC free article] [PubMed] [Google Scholar] 18.Shoseyov D., Bibi H., Biesalski H., Reifen R
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In the IUA model, this platform significantly repaired the activity and morphology of the endometrium by inhibiting fibrotic advancement markers (Lin et al., 2023)
