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Description
Acid-activated nonviral peptide vector for gene delivery
doi: 10.1165/ajrcmb.11.2.7519435 96 RanaldiG
This recessive disorder has stayed within the genotypes of humans most probably because the deficiency has been found to protect against a very lethal form of malaria
Inflammation, and autoimmunity
The Q-dsAMC probe reacted rapidly with NADH in the co-presence of DT-diaphorase and exhibited fluorescence activation (Fig
Moreover, replacement of Cys10 with serine in a TTR-V30M-based mouse model has been shown to prevent amyloid formation [24]
