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These pathways influence the activity of fusion proteins (dynamin-related protein 1) and segregation proteins (mitofusin 1/2 and optic atrophy type 1), as well as mitochondrial morphology [31]
These side effects are completely normal and typically resolve within a few days to a week
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Conversely, an abundance of arachidonic acid and other PUFAs can exacerbate the ferroptosis response when triggered by an inducer, as it accelerates the formation of peroxide lipids within the cell (40)
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