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In this classic signaling pathway, ligand-activated ER dissociates from its chaperone heat-shock protein and binds as a dimer either directly to an estrogen response element (ERE) in target gene promoters or indirectly to activator protein 1 or specificity protein 1 response elements through protein tethering to DNA (13)

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Their biosynthetic pathways are complex, unstable, and highly dynamic, and the lack of precise dosing windows and real-time monitoring technologies generates markedly uncertainty in therapeutic application [262]

doi: 10.1016/j.biomaterials.2011.06.073 50 GardnerAde Mingo PulidoRuffellB