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[DOI] [PubMed] [Google Scholar] 139.Ridaura V.K., Faith J.J., Rey F.E., Cheng J., Duncan A.E., Kau A.L., Griffin N.W., Lombard V., Henrissat B., Bain J.R., et al

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As emphasized before, these occlusion-like syndromes [19,24,27,29] shared the previously described peripheral and central deficits noted in the occlusion syndromes and were largely counteracted by the given BPC 157 therapy

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On the other hand, inhibition of GSH synthesis with BSO, independently of the cysteine pool, has been repeatedly described to induce ferroptosis ( via depletion of mitochondrial GSH pool leading to the release of cytochrome c, when combined with chemotherapy in breast cancer and leukemia, respectively (71, 72)

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While biological dressings can interact with the matrix in the wound bed to speed up the healing process, their use is limited due to decreased interaction with the wound tissue

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