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[DOI] [PubMed] [Google Scholar] 139.Ridaura V.K., Faith J.J., Rey F.E., Cheng J., Duncan A.E., Kau A.L., Griffin N.W., Lombard V., Henrissat B., Bain J.R., et al

As emphasized before, these occlusion-like syndromes [19,24,27,29] shared the previously described peripheral and central deficits noted in the occlusion syndromes and were largely counteracted by the given BPC 157 therapy

On the other hand, inhibition of GSH synthesis with BSO, independently of the cysteine pool, has been repeatedly described to induce ferroptosis ( via depletion of mitochondrial GSH pool leading to the release of cytochrome c, when combined with chemotherapy in breast cancer and leukemia, respectively (71, 72)

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While biological dressings can interact with the matrix in the wound bed to speed up the healing process, their use is limited due to decreased interaction with the wound tissue
