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Results The disruption of D2R-DAT stimulates locomotor activity As aforementioned, we developed an interfering peptide (TAT-DAT NT ) to disrupt the interaction between DAT and D2R, which can increase locomotor activity in mice [25]

After the injury, the BPC-157 peptide was administered intraperitoneally at doses of 10 g/kg and 10 ng/kg

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These safety differences stem from AOD-9604s mechanism as a C-terminal fragment lacking affinity for the traditional growth hormone receptor

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