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In schizophrenia (SZ), Down Syndrome, and bipolar disorder (BD), increased brain insulin resistance and reduced IGF-1 signaling drive allostatic load/overload, leading to physiological dysregulation and the presentation of more severe symptoms throughout the body [44,45,46]

This opposed the possibility that a generic response to D-HIS could explain the increased GSH level seen in the pGSH1-GAP1 strain with D-HIS

Combined, these data reveal new interactions between cellular redox homeostasis, secondary metabolism and metal ion homeostasis

However, none of these candidates could be confidently assigned as canonical catalase homologs based on sequence similarity criteria

These data demonstrate that RSSH and/or COS can provide protection when given at reperfusion in hearts subjected to I/R injury