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[ 347 ] BACH1 deficiency relieves 2,4,6trinitrobenzene sulfonic acid (TNBS)mediated colitis through upregulation of HO1 and M2 polarization antiinflammatory response in macrophages
Luminescence was measured with a GloMax 96 Microplate Luminometer (Promega)
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Net intestinal transport of oxalate reflects passive absorption and SLC26A6-mediated secretion
Viability was normalized to the corresponding DMSO control
A person with mold exposure may also have sinus congestion, chemical sensitivity, fatigue, poor stress tolerance, sleep disruption, and inflammatory flares
