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POSSIBLE ROLE OF NF-B EXPRESSION IN NEUROINFLAMMATION IN AUTISM The neuroinflammation in autism appears to be chronic and even excessive

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[ 42 ] RCC2, a mitotic regulator, stimulates glucose metabolism in glioma by increasing HK2 expression via BACH1dependent transcriptional mechanisms

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Analysis of responses of cad2 to virulent and avirulent strains of the oomycete Peronospora parasitica and the bacterium Pseudomonas syringae did not reveal compromised resistance (May et al., 1996b) but subsequent studies of cad2 and rax1-1 reported increased susceptibility to avirulent strains of P
