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Liao D, Liu X, Yuan X, et al

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Many studies in animal model systems show that dietary consumption of exogenous AGEs contribute to oxidative stress and inflammation which could contribute to a number of chronic disease states (reviewed in 27,28 )

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In contrast, the fat-5(-) mutants, in which only the synthesis of C16:1n-9 and C18:1n-7 MUFAs were blocked, were able to generate large LDs under the JUb74 diet (Figure 6D)

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